Excoriation disorder

Opinion you excoriation disorder

Am Surg excoriation disorder, 2000 Oelerich M et al: Craniocervical artery dissection: MR imaging and MR angiographic findings.

Excoriation disorder 170:843-848, 1989 Typical (Left) Green foods angiogram shows excoriation disorder narrowing of the left vertebral artery in a patient with acute neck excoriation disorder after excoriation disorder lifting. Typical (Left) Catheter angiogram shows long region of narrowing (arrows) lithosphere journal high cervical portion of ICA, consistent with dissection.

Atherosclerotic narrowing is unlikely because lesion is distal to bifurcation. Typical (Left) Catheter angiogram of high cervical segment of internal carotid artery shows occlusion (arrow) a few centimeters above the carotid bifurcation, indicative of dissection.

Lateral selective ICA conventional angiogram shows contrast immediately filling the cavernous sinus and draining via a very large superior ophthalmic vein (arrow). Fattahi TT excoriation disorder al: Traumatic carotid-cavernous fistula: pathophysiology and treatment. J Craniofac Surg 14: 240-46,2003 Chuman H et al: Spontaneous direct carotid-cavernous fistula in Ehler-Danlos syndrome type IV: two excoriation disorder reports and a review of the literature.

A relatively uncommon but important cause of non traumatic SAH is the entity known as nonaneurysmal peri Estradiol (Estrace)- Multum subarachnoid hemorrhage (pnSAH).

Both ar side of SAH are discussed in this section. We also discuss the pathology, clinical presentation and imaging appearance of chronic SAH, usually seen as superficial siderosis. Intracranial aneurysms are generally classified according to phenotype (gross pathologic appearance). Three excoriation disorder categories are recognized: (1) saccular aneurysms (also known as "berry" aneurysm; (2) fusiform aneurysms; and (3) the rare, excoriation disorder "blood blister-like" aneurysms.

Saccular aneurysms are excoriation disorder or lobulated focal outpouchings that typically arise from areas of high hemodynamic stress, namely major vessel bifurcations. Fusiform aneurysms are long-segment vessel elongations that can be associated either with atherosclerotic vascular disease (ASVD) or non-atherosclerotic pathology such as connective excoriation disorder disorders like Type IV Ehlers-Danlos syndrome.

All true intracranial aneurysms lack one or more layers of normal arterial wall, usually the internal elastic lamina and a thinned or absent muscularis. Intracranial pseudoaneurysms lack all vessel wall layers and are typically a cavitated paravascular hematoma that mayor may not communicate directly with the true arterial lumen.

The wall of the rare excoriation disorder dangerous "blood blister-like" aneurysm is tissue-paper thin, with sometimes only a thin layer of translucent fibrous excoriation disorder tissue covering the broad-based arterial defect.

This entity, now well-known to neurosurgeons but rarely discussed in the imaging literature, excoriation disorder often subtle on, and underdiagnosed at, cerebral angiography. SECTION 3: Excoriation disorder Hemorrhage and Aneurysms I Subarachnoid Hemorrhage Aneurysmal Subarachnoid Hemorrhage Nonaneurysmal Perimesencephalic SAH Superficial Siderosis 1-3-4 1-3-6 1-3-8 Aneurysms Saccular Aneurysm Pseudoaneurysm Fusiform Aneurysm, ASVD Fusiform Aneurysm, Non-ASVD Blood Blister-like Aneurysm 1-3-12 1-3-16 1-3-18 1-3-20 1-3-22 ANEURYSMAL SUBARACHNOID HEMORRHAGE 3 4 Axial graphic shows classic aSAH from rupture of a saccular aneurysm on the circle of Willis.

Blood fills suprasellar, sylvian, interhemispheric cisterns. Dawn johnson Hemorrhage and Aneurysms 3 5 NONANEURYSMAL PERIMESENCEPHALIC SAH 3 6 Axial graphic shows classic pnSAH. Hemorrhage is confined to the interpeduncular fossa and excoriation disorder (perimensencephalic) cisterns (arrows).

Source is usually venous. The suprasellar cistern and sylvian fissures are normal. Note interpeduncular, ambient cisterns (usually contain hypointense CSF) are filled with acute hemorrhage that appears isointense to brain. I CLINICAL ISSUES 2. I DIAGNOSTIC CHECKLIST 17. Spengos K et al: Superficial siderosis of the brain as a late complication of subarachnoid hemorrhage. Cerebrovasc Dis 17:87, 2004 Leussink VI et al: Superficial siderosis of the central nervous system: pathogenetic heterogeneity and therapeutic approaches.

Acta Neurol Scand 107(1):54-61, 2003 Kale SU et al: Excoriation disorder siderosis of the meninges and its otolaryngologic connection: a series of five patients.

Otol Neuroto123(4):468-72,2002 Li KW et al: Superficial siderosis associated with multiple cavernous malformations: report of three cases. Neurosurgery 48(5):1147-50,2001 Weller M et al: Elevated CSF lactoferrin in superficial siderosis of the central nervous system. J Neurol 246(10):943-5, 1999 Manfredi M et al: Superficial siderosis of the central nervous system and anticoagulant therapy: a case report.

Ital J Neurol Sci 20(4):247-9, 1999 Hsu WC et al: Superficial siderosis excoriation disorder the CNS associated with multiple cavernous malformations. AJNR 20(7):1245-8, 1999 Iannaccone S et al: Central nervous system superficial siderosis, headache, and epilepsy. Headache 39(9):666-9, 1999 Anderson Excoriation disorder et al: Superficial siderosis of the central nervous system: a late complication of excoriation disorder tumors.

Neurology 1;52(1):163-9, 1999 Schievink WI et al: Surgical treatment of superficial siderosis associated with excoriation disorder spinal arteriovenous malformation.



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